phrl-tk synthetic renilla luciferase reporter vector (Promega)
90
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Promega
phrl-tk synthetic renilla luciferase reporter vector
Phrl Tk Synthetic Renilla Luciferase Reporter Vector, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/phrl-tk+synthetic+renilla+luciferase+reporter+vector/pgl3+basic/pm16376453-76-1-10
Average 90 stars, based on 1 article reviews
Phrl Tk Synthetic Renilla Luciferase Reporter Vector, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/phrl-tk+synthetic+renilla+luciferase+reporter+vector/pgl3+basic/pm16376453-76-1-10
Average 90 stars, based on 1 article reviews
phrl-tk synthetic renilla luciferase reporter vector - by Bioz Stars,
2026-10
90/100 stars
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Luciferase:Article Title: COX-2 inhibitors suppress lung cancer cell growth by inducing p21 via COX-2 independent signals. Article Snippet: Lung Cancer (2006) 51, 283—296 COX-2 inhibitors suppress lung cancer cell growth by inducing p21 via COX-2 independent signals ShouWei Hana,∗, Jesse Romana,b a Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University School of Medicine, Whitehead Bioresearch Building, 615 Michael Street, Suite 205-M, Atlanta, GA 30322, USA b Atlanta Veterans Affairs Medical Center, Atlanta, GA 30033, USA Received 27 June 2005; received in revised form 17 October 2005; accepted 31 October 2005 KEYWORDS p21; COX-2 inhibitor; Human lung carcinoma cells; Spl; Erk Summary COX-2 has been implicated in the control of human non-small cell lung carcinoma (NSCLC) cell growth.. The mechanisms by which COX-2 exerts its mitogenic effects have not been entirely elucidated, but stimulation of prostaglandin E2 production and alterations in the expression of the cyclin-dependent kinase inhibitor p21WAF-1/CIP1/MDA-6(p2i) have been suggested.. Here, we demonstrate that two COX2 inhibitors (NS398 and Nimesulide) inhibit proliferation and induce apoptosis in NSCLC cells, and these effects were associated with induction of p21 mRNA and protein expression. Plasmid Preparation:Article Title: COX-2 inhibitors suppress lung cancer cell growth by inducing p21 via COX-2 independent signals. Article Snippet: Lung Cancer (2006) 51, 283—296 COX-2 inhibitors suppress lung cancer cell growth by inducing p21 via COX-2 independent signals ShouWei Hana,∗, Jesse Romana,b a Division of Pulmonary, Allergy and Critical Care Medicine, Department of Medicine, Emory University School of Medicine, Whitehead Bioresearch Building, 615 Michael Street, Suite 205-M, Atlanta, GA 30322, USA b Atlanta Veterans Affairs Medical Center, Atlanta, GA 30033, USA Received 27 June 2005; received in revised form 17 October 2005; accepted 31 October 2005 KEYWORDS p21; COX-2 inhibitor; Human lung carcinoma cells; Spl; Erk Summary COX-2 has been implicated in the control of human non-small cell lung carcinoma (NSCLC) cell growth.. The mechanisms by which COX-2 exerts its mitogenic effects have not been entirely elucidated, but stimulation of prostaglandin E2 production and alterations in the expression of the cyclin-dependent kinase inhibitor p21WAF-1/CIP1/MDA-6(p2i) have been suggested.. Here, we demonstrate that two COX2 inhibitors (NS398 and Nimesulide) inhibit proliferation and induce apoptosis in NSCLC cells, and these effects were associated with induction of p21 mRNA and protein expression. |